Author

S. M. Peirce

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 5 Approach:experimental assay· 4 Organism:mouse· 3 Research Focus:development & morphogenesis· 2 Research Focus:differential expression· 2 Assay:imaging· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

B. A. Corliss· 2 shared B. E. Isakson· 2 shared A. C. Bjork· 1 shared A. C. Bruce· 1 shared A. Miller· 1 shared A. S. Keller· 1 shared B. Corliss· 1 shared B. H. Corliss· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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2 shared papersPhysiology

B. A. Corliss

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Multiscale computational model predicts how environmental changes and drug treatments affect microvascular remodeling in fibrotic disease

J. Leonard-Duke, S. M. J. Agro, D. J. Csordas, A. C. Bruce, T. G. Eggertsen, T. N. Tavakol, T. H. Barker, C. A. Bonham, J. J. Saucerman, L. J. Taite, S. M. Peirce

The paper uses a multiscale computational model to predict how environmental changes and drug treatments affect microvascular remodeling in idiopathic pulmonary fibrosis.

bioRxiv · Mar 15, 2024Bioengineering

Polarized localization of phosphatidylserine in endothelium regulates Kir2.1

C. A. Ruddiman, R. Peckham, M. A. Luse, Y. L. A. Chen, M. Kuppusamy, B. Corliss, P. J. Hall, C.-J. Lin, S. M. Peirce, S. K. Sonkusare, R. P. Mecham, J. E. Wagenseil, B. E. Isakson

The paper shows phosphatidylserine (PS) localizes to myoendothelial junctions (MEJs) in endothelium, inhibiting PIP2-mediated Kir2.1 activation to regulate vascular diameter.

bioRxiv · Aug 1, 2022Physiology

Extracellular Matrix Remodeling Associated With Bleomycin-Induced Lung Injury Supports Pericyte-To-Myofibroblast Transition

R. HANNAN, A. Miller, R.-C. Hung, C. Sano, S. M. Peirce, T. Barker

This paper investigates how pericytes transition into myofibroblasts during lung injury, showing that fibronectin and vβ3 integrin engagement drive this process and may be targeted to reduce fibrosis.

bioRxiv · Nov 16, 2020Cell Biology

Vascular Expression of Hemoglobin Alpha in Antarctic Icefish Supports Iron Limitation as Novel Evolutionary Driver

B. A. Corliss, L. J. Delalio, T. C. S. Keller, A. S. Keller, D. A. Keller, B. H. Corliss, A. C. Bjork, J. M. Beers, S. M. Peirce, B. E. Isakson

The paper shows Hb-3f is expressed in icefish vascular endothelium, supporting iron minimization as an evolutionary driver for hemoglobin loss in Antarctic Icefish.

bioRxiv · Jul 19, 2019Physiology

Pericyte Bridges in Homeostasis and Hyperglycemia: Reconsidering Pericyte Dropout and Microvascular Structures

B. A. Corliss, H. C. Ray, R. Doty, C. Mathews, N. Sheybani, K. Fitzgerald, R. Prince, M. Kelly-Goss, W. L. Murfee, J. Chappell, G. Owens, P. A. Yates, S. M. Peirce

The paper reevaluates pericyte dropout in diabetic retinopathy, showing pericyte bridges are misclassified and highlighting their role in disease progression.

bioRxiv · Jul 16, 2019 · PreprintCell Biology