Author

S. S. Gross

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:metabolism & bioenergetics· 2 Research Focus:cell type annotation· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Halama· 2 shared K. Suhre· 2 shared Q. Chen· 2 shared A. Iskandarani· 1 shared A. M. Bhagwat· 1 shared B. D. Stein· 1 shared B. Ngo· 1 shared D. Wu· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersCell Biology

A. Halama

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Author profile →

3 papers

Recent·Most cited

Progesterone induces meiosis through two obligate co-receptors with PLA2 activity

N. Nader, L. Assaf, L. Zarif, A. Halama, S. Yadav, M. Dib, N. Attarwala, Q. Chen, K. Suhre, S. S. Gross, K. Machaca

This study identifies ABHD2 as an essential co-receptor for mPRβ to trigger meiosis via PLA2 activity in Xenopus oocytes.

bioRxiv · Sep 9, 2023Cell Biology

Non-conserved metabolic regulation by LKB1 distinguishes human and mouse lung adenocarcinoma

B. D. Stein, J. R. Ferrarone, E. E. Gardner, J. W. Chang, D. Wu, Q. Chen, P. E. Hollstein, M. Yuan, R. J. Liang, J. S. Coukos, M. Sindelar, B. Ngo, S. S. Gross, R. J. Shaw, J. M. Asara, , L. C. Cantley

This study identifies a metabolic divergence between human and mouse lung adenocarcinomas due to LKB1-dependent regulation of TPI1 phosphorylation, highlighting limitations of mouse models for studying human cancer.

bioRxiv · Oct 1, 2021 · PreprintCancer Biology

Accelerated lipid catabolism and autophagy are cancer survival mechanisms under inhibited glutaminolysis

A. Halama, M. Kulinski, S. S. Dib, S. B. Zaghlool, K. S. Siveen, A. Iskandarani, N. J. Satheesh, A. M. Bhagwat, S. Uddin, G. Kastenmueller, O. Elemento, S. S. Gross, K. Suhre

The paper shows that inhibiting glutaminolysis in cancer cells leads to lipid catabolism and autophagy, which promote survival, and that combined inhibition of both pathways can induce cell death.

bioRxiv · Jan 15, 2018Cancer Biology