Author

S. S. Kang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. C. Silva· 1 shared B. R. Conklin· 1 shared C. R. Simoneau· 1 shared D. A. Joy· 1 shared D. Otgonnamjil· 1 shared E. J. Lee· 1 shared G. N. Ramadoss· 1 shared H. Li· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

A. C. Silva

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2 papers

Recent·Most cited

Interaction between PSD 95 and TRPV4 through PDZ domain controls TRPV4's localization and activity.

S. S. Kang, E. J. Lee, K. Kim, D. Otgonnamjil, S. H. Shin

This paper identifies PSD95 as an interacting protein of TRPV4, showing that their interaction regulates TRPV4's localization and activity through the PDZ domain.

bioRxiv · Jun 30, 2023 · PreprintCell Biology

SARS-CoV-2 infection of human iPSC-derived cardiac cells predicts novel cytopathic features in hearts of COVID-19 patients

J. A. Perez-Bermejo, S. S. Kang, S. J. Rockwood, C. R. Simoneau, D. A. Joy, G. N. Ramadoss, A. C. Silva, W. R. Flanigan, H. Li, K. Nakamura, J. D. Whitman, M. Ott, B. R. Conklin, T. C. McDevitt

This study infects human iPSC-derived cardiac cells with SARS-CoV-2 and identifies novel cytopathic features in COVID-19 patient hearts, including myofibrillar fragmentation and DNA loss in cardiomyocytes.

bioRxiv · Aug 25, 2020Cell Biology