Author

S. Schaubeck

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

K. Husnjak· 2 shared A. Fornili· 1 shared A. Gubas· 1 shared A. Kalousi· 1 shared A. Pandini· 1 shared B. Stieglitz· 1 shared D. Kuntin· 1 shared F. Langschied· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiochemistry

K. Husnjak

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Author profile →

2 papers

Recent·Most cited

N4BP1 is dimerization-dependent linear ubiquitin reader regulating TNFR1 signalling through linear ubiquitin binding and Caspase-8-mediated processing

K. W. Kliza, W. Song, I. Pinzuti, S. Schaubeck, S. Kunzelmann, D. Kuntin, A. Fornili, A. Pandini, K. Hofmann, J. Garnett, B. Stieglitz, K. Husnjak

This study identifies N4BP1 as a dimerization-dependent linear ubiquitin reader that regulates TNFR1 signaling by modulating NF-κB and apoptosis pathways.

bioRxiv · Nov 2, 2021 · PreprintBiochemistry

Combinatorial CRISPR screening reveals functional buffering in autophagy

V. Diehl, M. Wegner, P. Grumati, K. Husnjak, S. Schaubeck, A. Gubas, V. J. Shah, F. Langschied, A. Kalousi, I. Ebersberger, I. Dikic, M. Kaulich

This study uses combinatorial CRISPR screening to identify gene interactions in autophagy, revealing functional buffering and redundant gene circuits.

bioRxiv · Jul 28, 2020Cell Biology