Author

S. Southekal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:multiome· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Guda· 2 shared C. Rao· 2 shared K. W. Fisher· 2 shared R. A. Svoboda· 2 shared R. E. Lewis· 2 shared A. R. Black· 1 shared D. Chatterjee· 1 shared D. E. Frodyma· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCell Biology

C. Guda

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Author profile →

2 papers

Recent·Most cited

Post-transcriptional control of SRSF9 promotes the epithelial-to-mesenchymal transition (EMT) in colorectal cancer cells

C. Rao, R. A. Svoboda, S. Southekal, H. M. Vieira, D. H. Huisman, D. Chatterjee, C. Guda, K. W. Fisher, O. Anczukow, R. E. Lewis

The paper shows that KSR1-dependent signaling in CRC cells promotes SRSF9-mediated splicing of EPSTI1 mRNA, which drives EMT and metastasis.

bioRxiv · May 16, 2022 · PreprintCell Biology

KSR1-and ERK-dependent Translational Regulation of the Epithelial-to-Mesenchymal Transition

C. Rao, D. E. Frodyma, S. Southekal, R. A. Svoboda, A. R. Black, C. Guda, T. Mizutani, H. Clever, K. R. Johnson, K. W. Fisher, R. E. Lewis

This study shows KSR1 and ERK promote EMT by selectively translating EPSTI1, which facilitates the E-to N-cadherin switch and enhances migration and invasion in CRC cells.

bioRxiv · Jan 18, 2021Cancer Biology