Author

S. Tagore

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Organism:E. coli· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

A. C. Iuga· 1 shared A. Califano· 1 shared A. Curiel Garcia· 1 shared A. Griffin· 1 shared A. L. E. Wang· 1 shared B. Schmierer· 1 shared C. Maurer· 1 shared D. Tuveson· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. C. Iuga

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2 papers

Recent·Most cited

tRNA Methylation Resolves Codon Usage Bias at the Limit of Cell Viability

I. Masuda, Y. Yamaki, R. Detroja, S. Tagore, H. Moore, S. Maharjan, Y. Nakano, T. Christian, R. Matsubara, T. M. Lowe, M. Frenkel-Morgenstern, Y.-M. Hou

This study shows that m1G37 tRNA modification neutralizes differential proline codon decoding, and its absence requires a minor isoacceptor for E. coli viability.

bioRxiv · May 14, 2022Microbiology

Developmental and MAPK-responsive transcription factors drive distinct malignant subtypes and genetic dependencies in pancreatic cancer

P. Laise, M. Turunen, A. Curiel Garcia, L. Tomassoni, C. Maurer, E. Elyada, B. Schmierer, J. Worley, J. Kesner, X. Tan, E. Calvo Fernandez, K. Wong, U. Wasko, S. Tagore, A. L. E. Wang, , A. Califano

The paper identifies three developmental lineages in pancreatic cancer driven by master regulators and shows their association with MAPK activity and therapeutic vulnerabilities.

bioRxiv · Oct 27, 2020 · PreprintCancer Biology