Author

S. Tumanov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

J. Nadel· 2 shared R. Stocker· 2 shared A. Baker· 1 shared A. Bongers· 1 shared A. Jabbour· 1 shared A. Lin· 1 shared A. Misra· 1 shared A. Muinonen-Martin· 1 shared

Activity by Year

2017-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersBiochemistry

J. Nadel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Myeloperoxidase activity predicts atherosclerotic plaque disruption and atherothrombosis

J. Nadel, X. Wang, P. Saha, A. Bongers, S. Tumanov, N. Giannotti, W. Chen, N. Vidger, M. M. Chowdhury, G. J. Lima da Cruz, C. Velasco, C. Prieto, A. Jabbour, R. M. Botnar, R. Stocker, A. Phinikaridou

This study shows that myeloperoxidase (MPO) activity in atherosclerotic plaques predicts atherothrombosis in rabbits and correlates with ruptured human atheroma.

bioRxiv · Oct 8, 2023 · PreprintBiochemistry

Colchicine promotes atherosclerotic plaque stability independently of inflammation

W. Li, A. Lin, M. Hutton, H. Dhaliwal, J. Nadel, J. Rodor, S. Tumanov, T. Örd, M. Hadden, M. Mokry, B. M. Mol, G. Pasterkamp, M. P. Padula, C. L. Geczy, Y. Ramaswamy, , A. Misra

This study shows colchicine stabilizes atherosclerotic plaques by converting harmful SMC-derived cells into protective myofibroblast-like cells, independent of its anti-inflammatory effects.

bioRxiv · Oct 3, 2023 · PreprintCell Biology

LPP3 mediates self-generation of chemotactic LPA gradients by melanoma cells

O. Susanto, Y. Koh, N. Morrice, S. Tumanov, P. Thomason, M. Neilson, L. Tweedy, A. Muinonen-Martin, J. Kamphorst, G. Mackay, R. H. Insall

The paper shows that LPP3 is essential for melanoma cells to break down LPA, enabling self-generated chemotaxis and invasion.

bioRxiv · Jun 21, 2017Cancer Biology