Author

Sahana Natarajan

Broad InstitutePrimary ORCID profile (0009-0002-0365-1151 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Sahana NatarajaniD1 paper · Boston University

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

Antia Valle-TojeiroiD· 3 shared Deeksha Misri· 3 shared Hasmik KeshishianiD· 3 shared Min Jee KwoniD· 3 shared Morgan ShengiD· 3 shared Sameer AryaliD· 3 shared Steven A. CarriD· 3 shared Ally A. NicolellaiD· 2 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Antia Valle-TojeiroORCID

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3 papers

Recent·Most cited

NeuroscienceNature CommunicationsPeer-reviewedNov 28, 2025

Elevated synaptic PKA activity and abnormal striatal dopamine signaling in Akap11 mutant mice, a genetic model of schizophrenia and bipolar disorder

Bryan J. Song, Yang Ge, Ally A. Nicolella…·Morgan Sheng

This study investigates Akap11 mutant mice to explore the neurobiological effects of AKAP11 loss, finding increased PKA activity and transcriptomic changes linked to schizophrenia and bipolar disorder.

17 citationstop 1%

mousebulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 26, 2025

The G-protein coupled receptor GPR34 maintains the homeostatic state of microglia and restrains the disease-associated microglial response in an Alzheimers disease model

Ana Geller, Min Jee Kwon, Sean Simmons…·Prabhat S. Kunwar

This study shows that GPR34 maintains microglial homeostasis and limits disease-associated microglial responses in Alzheimer's disease models.

1 citation

𝕏 PostBluesky

NeurosciencebioRxivPreprintOct 12, 2024

Muti-omics characterization reveals brain-wide disruption of synapses and region- and age-specific changes in neurons and glia in Sp4 mutant mice, a genetic model of schizophrenia and bipolar disorder

Min Jee Kwon, Kira A. Perzel Mandell, Sameer Aryal…·Morgan Sheng

This study uses multi-omics analyses of Sp4 mutant mice to reveal synapse disruption and age- and region-specific neuronal and glial changes linked to schizophrenia and bipolar disorder.

2 citations

mousebulk RNA-seq

𝕏 PostBluesky