Author

Ana Geller

Broad Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Hasmik KeshishianiD· 2 shared Katherine J. StalnakeriD· 2 shared Min Jee KwoniD· 2 shared Morgan ShengiD· 2 shared Sameer AryaliD· 2 shared Steven A. CarriD· 2 shared Ahmet S. AsaniD· 1 shared Alexandra E. GouldiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersNeuroscience

Hasmik KeshishianORCID

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2 papers

Recent·Most cited

The G-protein coupled receptor GPR34 maintains the homeostatic state of microglia and restrains the disease-associated microglial response in an Alzheimers disease model

Ana Geller, Min Jee Kwon, Sean Simmons, Qihong Xu, William E. Martenis, Jordan L. Doman, Sahana Natarajan, Katherine J. Stalnaker, Jixiang Zhang, Xian Adiconis, Trang Nguyen, Matthew Demers, Donovan L. Batzli, Antia Valle-Tojeiro, Christy Biji, , Prabhat S. Kunwar

This study shows that GPR34 maintains microglial homeostasis and limits disease-associated microglial responses in Alzheimer's disease models.

bioRxiv · Nov 26, 2025 · PreprintNeuroscience

SRRM2 haploinsufficiency drives SynGAP-γ reduction, Agap3 mis-splicing, and oligodendrocyte deficits in a mouse model of schizophrenia

Sameer Aryal, Chuhan Geng, Margaret A. Yin, Min Jee Kwon, Zohreh Farsi, Nathaniel Goble, Ahmet S. Asan, Kira S. Brenner, Nate Shepard, Ana Geller, Katherine J. Stalnaker, Olivia Seidel, Yining Wang, Ally A. Nicolella, Bryan J. Song, , Morgan Sheng

This study shows that SRRM2 haploinsufficiency in mice causes synaptic, splicing, and myelination defects linked to schizophrenia-like behaviors and gene expression changes.

bioRxiv · Oct 10, 2024 · PreprintNeuroscience