Author

Samuel A. Mills

Monash University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Bradley J. TurneriD· 2 shared David G. GonsalveziD· 2 shared Samantha K. BartoniD· 2 shared A. ColeiD· 1 shared Aida Viden· 1 shared Brittany CuiciD· 1 shared Catriona McLeaniD· 1 shared Doris TomasiD· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersNeuroscience

Bradley J. TurnerORCID

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2 papers

Recent·Most cited

A Human iPSC-derived Organoid System to Model Myelin Destruction and Repair

Shwathy Ramesan, Joel Mason, Molly E. V. Swanson, Samuel A. Mills, Bradley J. Turner, Matt Rutar, David G. Gonsalvez, Samantha K. Barton

This paper describes a human iPSC-derived organoid system that models myelin destruction and repair by incorporating mature oligodendrocytes and reactive microglia.

bioRxiv · May 19, 2024 · PreprintNeuroscience

Cortical hyperexcitability drives dying forward ALS symptoms and pathology in mice

Mouna Haidar, Aida Viden, Brittany Cuic, Taide Wang, Marius Rosier, Doris Tomas, Samuel A. Mills, A. Cole, Elvan Djouma, Sophia J. Luikinga, Valeria Rytova, Samantha K. Barton, David G. Gonsalvez, Lucy M. Palmer, Catriona McLean, , Bradley J. Turner

This study shows that cortical hyperexcitability in mice leads to ALS-like motor symptoms and pathology through a dying-forward mechanism.

bioRxiv · Aug 13, 2021 · PreprintNeuroscience