Author

Marius Rosier

The University of MelbourneORCID 0000-0002-9732-5543Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:imaging· 2 Assay:electronic health records· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Lucy M. PalmeriD· 4 shared Tomás J. RyaniD· 2 shared A. ColeiD· 1 shared Aida Viden· 1 shared Ana Morello Megias· 1 shared Ann-Sofie BjerreiD· 1 shared Bradley J. TurneriD· 1 shared Brittany CuiciD· 1 shared

Activity by Year

2021-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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4 shared papersNeuroscience

Lucy M. PalmerORCID

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4 papers

Recent·Most cited

Learning-induced plasticity decreases cortical engram cell dendritic excitability during memory recall

Marius Rosier, George Stuyt, Luca Godenzini, Tomás J. Ryan, Lucy M. Palmer

This study shows that engram cells in the auditory cortex have reduced dendritic excitability during memory recall, mediated by increased Ih current.

bioRxiv · Jan 8, 2025 · PreprintNeuroscience

Higher order thalamus encodes correct goal-directed action

Danilo La Terra, Ann-Sofie Bjerre, Marius Rosier, Rei Masuda, Tomás J. Ryan, Lucy M. Palmer

This study investigates the role of thalamic axons in goal-directed behavior, showing they encode correct actions and inhibit cortical neurons.

eLife · Mar 8, 2022Peer-reviewedNeuroscience

Higher synaptic threshold for NMDA spike generation in human neurons

Guilherme Testa-Silva, Marius Rosier, Suraj Honnuraiah, Robertas Guzulaitis, Ana Morello Megias, Chris R. French, James A. J. King, Katharine Jann Drummond, Lucy M. Palmer, Greg J. Stuart

The paper finds that human neurons have a higher synaptic threshold for NMDA spike generation compared to rodent neurons, primarily due to larger dendrite diameter.

bioRxiv · Oct 22, 2021 · PreprintNeuroscience

Cortical hyperexcitability drives dying forward ALS symptoms and pathology in mice

Mouna Haidar, Aida Viden, Brittany Cuic, Taide Wang, Marius Rosier, Doris Tomas, Samuel A. Mills, A. Cole, Elvan Djouma, Sophia J. Luikinga, Valeria Rytova, Samantha K. Barton, David G. Gonsalvez, Lucy M. Palmer, Catriona McLean, , Bradley J. Turner

This study shows that cortical hyperexcitability in mice leads to ALS-like motor symptoms and pathology through a dying-forward mechanism.

bioRxiv · Aug 13, 2021 · PreprintNeuroscience