Samantha K. Barton

This study shows that rescuing Schwann cells in an ALS mouse model improves myelin structure but not motor function, highlighting the complex role of Schwann cells in disease progression.

This paper describes a human iPSC-derived organoid system that models myelin destruction and repair by incorporating mature oligodendrocytes and reactive microglia.

This study shows that cortical hyperexcitability in mice leads to ALS-like motor symptoms and pathology through a dying-forward mechanism.

The paper investigates the impact of a t(1;11) translocation on white matter integrity and oligodendrocyte-myelin function using DTI and cellular models.