Author

Sara Al Rawi

University of Cambridge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Organism:human· 1

Frequent Co-Authors

Heike LamaniD· 3 shared Roger A. BarkeriD· 2 shared Antara MajumdariD· 1 shared Benjamin Matthew SkinneriD· 1 shared Claudia Cattoni Rathje· 1 shared David E. NelsoniD· 1 shared Emma Elizabeth Philippa JohnsoniD· 1 shared Joanne Bacon· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersNeuroscience

Heike LamanORCID

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Author profile →

3 papers

Recent·Most cited

Restoration of Fbxo7 expression in dopaminergic neurons restores tyrosine hydroxylase in a mouse model of Parkinson's disease

Sara Al Rawi, Pamela Tyers, Roger A. Barker, Heike Laman

This study shows that restoring Fbxo7 expression in dopaminergic neurons of a mouse Parkinson's model rescues tyrosine hydroxylase levels in the striatum and nucleus accumbens.

bioRxiv · Sep 28, 2024 · PreprintNeuroscience

Study of an FBXO7 patient mutation reveals Fbxo7 and PI31 co-regulate proteasomes and mitochondria

Sara Al Rawi, Lorna Simpson, Neil Q. McDonald, Veronika Chernuha, Orly N Elpeleg, Massimo Zeviani, Roger A. Barker, Ronen Spiegel, Heike Laman

This study identifies an FBXO7 mutation that disrupts its interaction with PI31, affecting proteasome and mitochondrial function in patient cells.

bioRxiv · Dec 22, 2021 · PreprintNeuroscience

A conserved requirement for Fbxo7 during male germ cell cytoplasmic remodelling

Claudia Cattoni Rathje, Suzanne J. Randle, Sara Al Rawi, Benjamin Matthew Skinner, David E. Nelson, Antara Majumdar, Emma Elizabeth Philippa Johnson, Joanne Bacon, Myrto Vlazaki, Nabeel Ahmed Affara, Peter J. I. Ellis, Heike Laman

This paper shows that Fbxo7 is required for male fertility in mice, as its deficiency leads to sterile males with abnormal spermatid disposal during spermiogenesis.

Frontiers in Physiology · Oct 10, 2019Peer-reviewedDevelopmental Biology