Author

David E. Nelson

Middle Tennessee State UniversityORCID 0000-0002-5034-0885Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Todd Davis· 1 shared Andrew EnglandiD· 1 shared Andrew J. OliveiD· 1 shared Antara MajumdariD· 1 shared Benjamin Matthew SkinneriD· 1 shared Cathrine A. Hasan· 1 shared Cedra H. Kamel· 1 shared Claudia Cattoni Rathje· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. Todd Davis

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Intracellular C. neoformans infection stimulates increased glycolytic activity in fetal liver-derived alveolar-like macrophages

Derek A. Wiggins, Josh B. Griggs, Andrew England, Emily N. Callison, Cedra H. Kamel, Cathrine A. Hasan, A. Todd Davis, Morgan E. Chappell, Kayla N. Conner, Andrew J. Olive, Erin E. McClelland, Rachel N. Leander, Rebecca L. Seipelt-Thiemann, David E. Nelson

This study shows that C. neoformans infection increases glycolytic activity in fetal liver-derived alveolar-like macrophages, which is HIF1-dependent.

bioRxiv · Nov 28, 2025 · PreprintImmunology

A conserved requirement for Fbxo7 during male germ cell cytoplasmic remodelling

Claudia Cattoni Rathje, Suzanne J. Randle, Sara Al Rawi, Benjamin Matthew Skinner, David E. Nelson, Antara Majumdar, Emma Elizabeth Philippa Johnson, Joanne Bacon, Myrto Vlazaki, Nabeel Ahmed Affara, Peter J. I. Ellis, Heike Laman

This paper shows that Fbxo7 is required for male fertility in mice, as its deficiency leads to sterile males with abnormal spermatid disposal during spermiogenesis.

Frontiers in Physiology · Oct 10, 2019Peer-reviewedDevelopmental Biology