Author

Simone Backes

University of Würzburg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioinformatics· 1 Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Adam W. WhisnantiD· 1 shared Anne-Sophie Gribling-BurreriD· 1 shared Caroline C. FriedeliD· 1 shared Gerti BeliuiD· 1 shared Jianhui LiiD· 1 shared Joél Schaust· 1 shared Lara DjakovićiD· 1 shared Lars DölkeniD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperBioinformatics

Adam W. WhisnantORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Visualizing the transcription and replication of influenza A viral RNAs in cells by multiple direct RNA padlock probing and in-situ sequencing (mudRapp-seq)

Shazeb Ahmad, Jianhui Li, Joél Schaust, Anne-Sophie Gribling-Burrer, Nina Geiger, Sabine Fischer, Uddhav B. Ambi, Simone Backes, Markus Johannes Ankenbrand, Redmond P. Smyth

This study introduces mudRapp-seq to visualize influenza A viral RNA transcription and replication at the single-cell level, revealing early mRNA expression and differential segment expression.

Nucleic Acids Research · May 23, 2025Peer-reviewedMicrobiology

Coronaviruses use ACE2 monomers as entry receptors

Patrick Eiring, Teresa Klein, Simone Backes, Marcel Streit, Marvin Jungblut, Sören Doose, Gerti Beliu, Markus Sauer

This study shows that SARS-CoV-2 uses monomeric ACE2 receptors for infection, with single S protein interactions sufficient for entry.

Angewandte Chemie International Edition · Mar 27, 2023Peer-reviewedMolecular Biology

HSV-1 and influenza infection induce linear and circular splicing of the long NEAT1 isoform

Marie-Sophie Friedl, Lara Djaković, Michael Kluge, Thomas Hennig, Adam W. Whisnant, Simone Backes, Lars Dölken, Caroline C. Friedel

The paper shows HSV-1 and influenza infection induce circular and linear splicing of the NEAT1_2 isoform, independent of vhs and linked to transcription termination disruption.

PLoS ONE · Oct 24, 2022Peer-reviewedBioinformatics