Author

Soad Elziny

University of Maryland, Baltimore

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Peter B. CrinoiD· 2 shared Philip H. IfflandiD· 2 shared Alexandros PoulopoulosiD· 1 shared Allan E. Barnes· 1 shared Amanda RinglandiD· 1 shared Andrea J. RomanowskiiD· 1 shared Anna Lanier· 1 shared Anoj IlangesiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

Peter B. CrinoORCID

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Author profile →

3 papers

Recent·Most cited

Reconstructing a physiological state space via chronic jugular microdialysis in freely moving mice

Michele Nardin, Nan Wang, Soad Elziny, Claire Boyer, Vojko Pjanovic, Luisa Schuster, Peter Boklund, Sarah E. Lindo, Kendra Morris, Anoj Ilanges, Jakob Voigts, Emily Jane Dennis

The paper measures hundreds of amines and peptides in freely moving mice using microdialysis and mass spectrometry, revealing high-dimensional physiological dynamics and state transitions.

bioRxiv · Dec 11, 2025 · PreprintPhysiology

Loss of Slc35a2 alters development of the mouse cerebral cortex

Soad Elziny, Sahibjot Sran, Hyojung Yoon, Rachel R. Corrigan, John Page, Amanda Ringland, Anna Lanier, Sara Lapidus, James Foreman, Erin L. Heinzen, Philip H. Iffland, Peter B. Crino, Tracy A. Bedrosian

This study shows that disrupting Slc35a2 in mouse cortical neurons causes altered neuronal migration and reduced seizure threshold, contributing to MOGHE-like features.

Neuroscience Letters · Jun 22, 2024Peer-reviewedNeuroscience

NPRL3: Direct Effects on Human Phenotypic Variability, mTOR Signaling, Subcellular mTOR Localization, Cortical Lamination, and Seizure Susceptibility

Philip H. Iffland, Mariah E. Everett, Katherine Cobb-Pitstick, Lauren E. Bowser, Allan E. Barnes, Janice K. Babus, Andrea J. Romanowski, Marianna Baybis, Soad Elziny, Erik G. Puffenberger, Claudia G. Gonzaga-Jauregui, Alexandros Poulopoulos, Vincent J. Carson, Peter B. Crino

The paper investigates NPRL3 variants' effects on mTOR signaling, cortical development, and epilepsy by using CRISPR/Cas9 knockout in cell lines and mice, showing mTOR hyperactivation and cortical dyslamination.

Brain · Feb 5, 2022Peer-reviewedNeuroscience