Author

John Page

Brown University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexander JaworskiiD· 1 shared Alexandra Weiss· 1 shared Alper KüçükuraliD· 1 shared Amanda RinglandiD· 1 shared Anna Lanier· 1 shared Diane LipscombeiD· 1 shared Emma M. ShychuckiD· 1 shared Erin L. HeinzeniD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Alexander JaworskiORCID

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2 papers

Recent·Most cited

Loss of Slc35a2 alters development of the mouse cerebral cortex

Soad Elziny, Sahibjot Sran, Hyojung Yoon, Rachel R. Corrigan, John Page, Amanda Ringland, Anna Lanier, Sara Lapidus, James Foreman, Erin L. Heinzen, Philip H. Iffland, Peter B. Crino, Tracy A. Bedrosian

This study shows that disrupting Slc35a2 in mouse cortical neurons causes altered neuronal migration and reduced seizure threshold, contributing to MOGHE-like features.

Neuroscience Letters · Jun 22, 2024Peer-reviewedNeuroscience

Up-regulation of cholesterol synthesis pathways and limited neurodegeneration in a knock-in Sod1 mutant mouse model of ALS.

Janice A. Dominov, Laura A. Madigan, Joshua P. Whitt, Katerina Rademacher, Kristin M. Webster, Hesheng Zhang, Haruhiko Banno, Siqi Tang, Yifan Zhang, Nicholas Wightman, Emma M. Shychuck, John Page, Alexandra Weiss, Karen L. Kelly, Alper Küçükural, , Robert H. Brown

This study shows that Sod1G85R knock-in mice exhibit up-regulated cholesterol synthesis pathways and limited neurodegeneration, contrasting with severe TgSOD1G93A mice, suggesting lipid pathway dysregulation in ALS.

bioRxiv · May 5, 2023 · PreprintNeuroscience