Author

Stefan J. Kempf

University of Southern Denmark

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Organism:mouse· 1 Assay:spatial transcriptomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Anne Marlene LandauiD· 1 shared Athanasios MetaxasiD· 1 shared Bente R. FinseniD· 1 shared Camilla ThygeseniD· 1 shared David James BrooksiD· 1 shared Hélène Audrain· 1 shared Jessica L. TeelingiD· 1 shared M AnzaloneiD· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Anne Marlene LandauORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Tauopathy in the APPswe/PS1ΔE9 mouse model of familial Alzheimer’s disease

Athanasios Metaxas, Camilla Thygesen, Stefan J. Kempf, M Anzalone, Ramanan Vaitheeswaran, Sussanne Petersen, Anne Marlene Landau, Hélène Audrain, Jessica L. Teeling, Sultan Darvesh, David James Brooks, Martin R. Larsen, Bente R. Finsen

This study shows that tau pathology develops spontaneously in APPswe/PS1ΔE9 mice, demonstrating that amyloidosis is necessary and sufficient to drive tauopathy in familial AD models.

Scientific Reports · Oct 31, 2019Peer-reviewedNeuroscience