Author

Ramanan Vaitheeswaran

University of Southern Denmark

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:imaging· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Athanasios MetaxasiD· 2 shared David James BrooksiD· 2 shared Hélène Audrain· 2 shared Marco AnzaloneiD· 2 shared Anne M. Landau· 1 shared Anne Marlene LandauiD· 1 shared Bente Finsen· 1 shared Bente R. FinseniD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersNeuroscience

Athanasios MetaxasORCID

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2 papers

Recent·Most cited

Age-specific Aβ-Tau interactions underlie spatial memory deficits in an APP/PS1 amyloidosis model

Athanasios Metaxas, Marco Anzalone, Ramanan Vaitheeswaran, Vivien Tejsi, Shuoshuo Li, Georgia Mitilineou, Kate L. Lambertsen, Hélène Audrain, David James Brooks, Anne M. Landau, Bente Finsen

This study examines how Aβ and tau interactions affect spatial memory in APP/PS1 mice, finding that memory decline is driven by combined Aβ and tau pathologies.

bioRxiv · Dec 10, 2025 · PreprintNeuroscience

Tauopathy in the APPswe/PS1ΔE9 mouse model of familial Alzheimer’s disease

Athanasios Metaxas, Camilla Thygesen, Stefan J. Kempf, M Anzalone, Ramanan Vaitheeswaran, Sussanne Petersen, Anne Marlene Landau, Hélène Audrain, Jessica L. Teeling, Sultan Darvesh, David James Brooks, Martin R. Larsen, Bente R. Finsen

This study shows that tau pathology develops spontaneously in APPswe/PS1ΔE9 mice, demonstrating that amyloidosis is necessary and sufficient to drive tauopathy in familial AD models.

Scientific Reports · Oct 31, 2019Peer-reviewedNeuroscience