Author

Sussanne Petersen

University of Southern Denmark

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Anne Marlene LandauiD· 2 shared Athanasios MetaxasiD· 2 shared Bente R. FinseniD· 2 shared M AnzaloneiD· 2 shared Camilla ThygeseniD· 1 shared David James BrooksiD· 1 shared Hélène Audrain· 1 shared Jessica L. TeelingiD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Anne Marlene LandauORCID

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2 papers

Recent·Most cited

Serotonin-2B receptor (5-HT2BR) expression and binding in the brain of ageing APPswe/PS1de9 transgenic mice and in human Alzheimer's disease brain tissue

M Anzalone, Sarmad A. Karam, Sanne R. R. Briting, Sussanne Petersen, Majken Borup Thomsen, Anne Marlene Landau, Bente R. Finsen, Athanasios Metaxas

This study examines 5-HT2BR expression and binding in AD mouse models and human tissue, finding age-related changes in mice and higher levels in AD patients.

bioRxiv · May 30, 2024 · PreprintNeuroscience

Tauopathy in the APPswe/PS1ΔE9 mouse model of familial Alzheimer’s disease

Athanasios Metaxas, Camilla Thygesen, Stefan J. Kempf, M Anzalone, Ramanan Vaitheeswaran, Sussanne Petersen, Anne Marlene Landau, Hélène Audrain, Jessica L. Teeling, Sultan Darvesh, David James Brooks, Martin R. Larsen, Bente R. Finsen

This study shows that tau pathology develops spontaneously in APPswe/PS1ΔE9 mice, demonstrating that amyloidosis is necessary and sufficient to drive tauopathy in familial AD models.

Scientific Reports · Oct 31, 2019Peer-reviewedNeuroscience