Author

Syed Haider

City College of New YorkPrimary ORCID profile (0000-0001-6685-5480 · 4 papers)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Syed HaideriD4 papers · Institute of Cancer Research

Biology Disciplines

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Cancer Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Ayesha Chowdhury· 1 shared Caroline Sproat· 1 shared Dean Hammond· 1 shared Gareth J. ThomasiD· 1 shared Horacio J. Maldonado-Lorca· 1 shared Ian A. PrioriD· 1 shared Itzhak ManoiD· 1 shared Jessica L. BeckeriD· 1 shared

Activity by Year

2018-2018 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Ayesha Chowdhury

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2 papers

Recent·Most cited

Non-Canonical Activation of CREB Mediates Neuroprotection in a C. elegans Model of Excitotoxic Necrosis

K. Genevieve Feldmann, Ayesha Chowdhury, Jessica L. Becker, N’Gina McAlpin, Taqwa Ahmed, Syed Haider, Jian X. Richard Xia, Karina Diaz, Monal Mehta, Itzhak Mano

The paper identifies a non-canonical CREB activation pathway via CRTC in C. elegans excitotoxic necrosis, showing it mediates neuroprotection independently of CREB phosphorylation.

Journal of Neurochemistry · Nov 17, 2018Peer-reviewedNeuroscience

Integrin αVβ6-EGFR crosstalk regulates bidirectional force transmission and controls breast cancer invasion

Joanna R. Thomas, Kate M. Moore, Caroline Sproat, Horacio J. Maldonado-Lorca, Stephanie Mo, Syed Haider, Dean Hammond, Gareth J. Thomas, Ian A. Prior, Pedro R. Cutillas, Louise J. Jones, John F. Marshall, Mark R. Morgan

This study shows that Integrin αVβ6-EGFR crosstalk regulates bidirectional force transmission and drives breast cancer invasion by coordinating matrix stiffness and transcriptional reprogramming.

bioRxiv · Sep 4, 2018 · PreprintCancer Biology