Author

T. Iwatsubo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

T. HASHIMOTO· 2 shared A. IKEDA· 1 shared G. Litscher· 1 shared H. An· 1 shared K. MASATO· 1 shared N. KANESHIRO· 1 shared N. TAKASUGI· 1 shared N. Watanabe· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiochemistry

T. HASHIMOTO

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Lipid flippase dysfunction as a novel therapeutic target for endosomal anomalies in Alzheimer's disease

N. KANESHIRO, K. MASATO, R. IMAOKA, A. IKEDA, Y. KAMIKUBO, T. HASHIMOTO, T. IWATSUBO, T. SAKURAI, T. UEHARA, N. TAKASUGI

This study identifies lipid flippase dysfunction as a key pathogenic event in Alzheimer's disease and presents a peptide therapy to restore endosomal function.

bioRxiv · Jul 30, 2021Biochemistry

Compositional analysis of ALS-linked stress granule-like structures reveals factors and cellular pathways dysregulated by mutant FUS under stress

H. An, G. Litscher, W. Wei, N. Watanabe, T. Hashimoto, T. Iwatsubo, V. L. Buchman, T. A. Shelkovnikova

This study compares the composition of normal stress granules and mutant FUS assemblies in stressed cells, identifying molecular differences and their impact on cellular stress responses in ALS-FUS.

bioRxiv · Mar 2, 2021 · PreprintCell Biology