Author

T. J. Weiss

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:DNA methylation· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

B. M. Murphy· 2 shared C. E. Burd· 2 shared C. J. Burd· 2 shared A. Dhakal· 1 shared A. H. Shain· 1 shared A. M. Holderbaum· 1 shared A. Webb· 1 shared D. A. Tallman· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

B. M. Murphy

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2 papers

Recent·Most cited

Functional differences drive the selection of NRAS mutants in melanoma

B. M. Murphy, T. J. Weiss, A. M. Holderbaum, A. Dhakal, M. Fort, M. S. Bodnar, M. Chen, C. J. Burd, V. COPPOLA, C. E. Burd

This study shows that specific NRAS mutants drive melanoma formation in mice, with Q61R and Q61K being the most effective, while others like G12D or Q61Q are not.

bioRxiv · Jan 15, 2021 · PreprintCancer Biology

UVB mutagenesis differs in NRAS- and BRAF-mutant mouse models of melanoma

R. L. Bowman, R. C. Hennessey, D. A. Tallman, T. J. Weiss, E. R. Crawford, B. M. Murphy, A. Webb, S. Zhang, K. M. D. La Perle, C. J. Burd, R. L. Levine, A. H. Shain, C. E. Burd

This study compares UVB mutagenesis effects in NRAS- and BRAF-mutant mouse melanoma models, finding BRAF-mutant tumors have more mutations and a stronger UVB mutational signature.

bioRxiv · Dec 27, 2020 · PreprintCancer Biology