Author

T. M. Molenaar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Organism:human· 1

Frequent Co-Authors

F. van Leeuwen· 2 shared C. Lancini· 1 shared C. McLean· 1 shared E. M. Kwesi-Maliepaard· 1 shared H. Jacobs· 1 shared H. Vlaming· 1 shared J. Silva· 1 shared J.-H. Dannenberg· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

F. van Leeuwen

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2 papers

Recent·Most cited

SETD2 negatively regulates cell size through its catalytic activity and SRI domain

T. M. Molenaar, E. M. Kwesi-Maliepaard, J. Silva, M. Malik, W. J. Faller, F. van Leeuwen

This study identifies SETD2 as a negative regulator of cell size through its catalytic activity, independent of H3K36 methylation, and shows that its SRI domain interacts with non-histone substrates to modulate protein synthesis.

bioRxiv · Oct 31, 2021Cell Biology

Evolutionarily-conserved chromatin crosstalk generates a DOT1L-dose dependency in thymic lymphoma caused by loss of HDAC1

H. Vlaming, C. McLean, T. Korthout, M. F. Alemdehy, S. Henriks, C. Lancini, S. Palit, S. Klarenbeek, T. M. Molenaar, L. Hoekman, T. T. Schmidlin, M. Altelaar, T. v. Welsem, J.-H. Dannenberg, H. Jacobs, F. van Leeuwen

The paper identifies an evolutionarily-conserved interaction between HDAC1 and DOT1L that influences H3K79 methylation and contributes to thymic lymphoma development.

bioRxiv · Jan 10, 2019Cell Biology