Author

T. M. Svitkina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1 Organism:mouse· 1

Frequent Co-Authors

A. T. Weeraratna· 1 shared A. Y. Shih· 1 shared B. Calabrese· 1 shared E. McEvoy· 1 shared E. Moeendarbary· 1 shared F. Spill· 1 shared G. E. Marino· 1 shared H. N. Higgs· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBiophysics

A. T. Weeraratna

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2 papers

Recent·Most cited

Cell BiologybioRxivOct 1, 2021

INF2-mediated actin filament reorganization confers intrinsic resilience to neuronal ischemic injury

B. Calabrese, S. Jones, Y. Yamaguchi-Shiraishi…·S. Halpain

This study shows that INF2-mediated actin reorganization in neurons protects against ischemic injury by counteracting swelling-induced cell death.

DOI 10.1038/s41467-022-33268-y

mouseexperimental assay

BiophysicsbioRxivPreprintMar 15, 2021

Modeling the three-way feedback between cellular contractility, actin polymerization, and adhesion turnover resolves the contradictory effects of RhoA and Rac1 on endothelial junction dynamics

E. McEvoy, T. Sneh, E. Moeendarbary…·V. B. Shenoy

This study develops a chemo-mechanical model to explain how RhoA and Rac1 differentially affect endothelial junction dynamics through their roles in contractility, adhesion turnover, and actin polymerization.

DOI 10.1101/2021.03.15.435512

humanmechanistic / dynamical model