Author

T. Sneh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biophysics· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Approach:mechanistic / dynamical model· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Genna· 2 shared H. Gil-Henn· 2 shared T. Meirson· 2 shared A. T. Weeraratna· 1 shared C. Sokolik· 1 shared D. Cox· 1 shared D. Entenberg· 1 shared E. McEvoy· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

A. Genna

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

FAK family proteins regulate in vivo breast cancer metastasis via distinct mechanisms.

A. Genna, J. Alter, M. Poletti, T. Meirson, T. Sneh, M. Gendler, N. Saleev, G. Karagiannis, Y. Wang, D. Cox, D. Entenberg, M. Oktay, T. Korcsmaros, J. Condeelis, H. Gil-Henn

This study shows that PYK2 and FAK regulate breast cancer metastasis through distinct mechanisms, with PYK2 affecting motility and FAK reducing tumor size and metastasis.

bioRxiv · Oct 27, 2023 · PreprintCancer Biology

A novel Pyk2-derived peptide inhibits invadopodia-mediated breast cancer metastasis

S. Twafra, C. Sokolik, T. Sneh, K. Srikanth, T. Meirson, A. Genna, J. Chill, H. Gil-Henn

This study identifies a Pyk2-derived peptide that inhibits invadopodia-mediated breast cancer metastasis by blocking cortactin interaction.

bioRxiv · Apr 6, 2022Cancer Biology

Modeling the three-way feedback between cellular contractility, actin polymerization, and adhesion turnover resolves the contradictory effects of RhoA and Rac1 on endothelial junction dynamics

E. McEvoy, T. Sneh, E. Moeendarbary, G. E. Marino, X. Chen, J. Escribano, F. Spill, J. M. Garcia-Aznar, A. T. Weeraratna, T. M. Svitkina, R. D. Kamm, V. B. Shenoy

This study develops a chemo-mechanical model to explain how RhoA and Rac1 differentially affect endothelial junction dynamics through their roles in contractility, adhesion turnover, and actin polymerization.

bioRxiv · Mar 15, 2021 · PreprintBiophysics