Author

T. Mito

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. E. Vincent· 1 shared A. Marmyleva· 1 shared A. Suomalainen-Wartiovaara· 1 shared C. B. Jackson· 1 shared C. J. Carroll· 1 shared E. Ogasawara· 1 shared G. Monteuuis· 1 shared J.-I. Hayashi· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. E. Vincent

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Author profile →

2 papers

Recent·Most cited

De novo serine biosynthesis is protective in mitochondrial disease

C. B. Jackson, A. Marmyleva, R. Awadhpersad, G. Monteuuis, T. Mito, N. Zamboni, T. Tatsuta, A. E. Vincent, L. Wang, T. Langer, C. J. Carroll, A. Suomalainen-Wartiovaara

This study shows that de novo serine biosynthesis protects against mitochondrial disease by maintaining metabolic homeostasis, redox balance, and lipid homeostasis in skeletal muscle.

bioRxiv · Mar 23, 2023 · PreprintCell Biology

Diabetic conditions induce intolerance to accumulation of pathogenic mitochondrial DNAs

E. Ogasawara, K. Nakada, S. Katada, T. Mito, J.-I. Hayashi

The paper shows diabetic conditions exacerbate mitochondrial respiration defects caused by pathogenic mtDNA mutations, suggesting diabetes may worsen mitochondrial diseases.

bioRxiv · Oct 2, 2019 · PreprintCell Biology