Author

Timothy P. Stinear

Primary ORCID profile (0000-0003-0150-123X · 4 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Timothy P. StineariD4 papers · The University of Melbourne

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:bacteria (other)· 1 Assay:DNA methylation· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Benjamin P. HowdeniD· 2 shared Jessica L. PorteriD· 2 shared Paul D. R. JohnsoniD· 2 shared Torsten SeemanniD· 2 shared Anders Gonçalves da SilvaiD· 1 shared Andrew James Stewardson· 1 shared Brian Howden· 1 shared Deborah A. Williamson· 1 shared

Activity by Year

2016-2017 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersMicrobiology

Benjamin P. HowdenORCID

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Author profile →

3 papers

Recent·Most cited

A target-specific assay for rapid and quantitative detection of Mycobacterium chimaera DNA in environmental and clinical specimens

Enrique Zozaya-Valdés, Jessica L. Porter, John Coventry, Janet A. M. Fyfe, Glen P. Carter, Anders Gonçalves da Silva, Torsten Seemann, Paul D. R. Johnson, Andrew James Stewardson, Ivan Bastian, Sally A. Roberts, Benjamin P. Howden, Deborah A. Williamson, Timothy P. Stinear

The paper develops a TaqMan qPCR assay for specific and rapid detection of M. chimaera DNA in clinical and environmental samples, showing higher sensitivity than culture methods.

bioRxiv · Feb 9, 2017 · PreprintMicrobiology

Refutation: Spontaneous secondary mutations confound analysis of the essential two component system WalKR in Staphylococcus aureus

Ian R. Monk, Torsten Seemann, Benjamin P. Howden, Timothy P. Stinear

The paper refutes claims about the essentiality of specific amino acid residues in the WalK sensor kinase of Staphylococcus aureus by showing that observed phenotypes may arise from secondary mutations rather than the intended single amino acid substitutions.

Nature Communications · Feb 6, 2017Peer-reviewedMicrobiology

Mycobacterium ulcerans low infectious dose and atypical mechanical transmission support insect bites and puncturing injuries in the spread of Buruli ulcer.

John R. Wallace, Kirstie M. Mangas, Jessica L. Porter, Renee Marcsisin, Sacha J. Pidot, Brian Howden, Till F. Omansen, Weiguang Zeng, Jason K. Axford, Paul D. R. Johnson, Timothy P. Stinear

The paper demonstrates that a low dose of Mycobacterium ulcerans can cause Buruli ulcer through skin punctures from mosquito bites or needles in mice.

bioRxiv · Aug 27, 2016 · PreprintMicrobiology