Author

U. Mayer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Research Focus:development & morphogenesis· 3 Approach:experimental assay· 3 Organism:Arabidopsis· 1 Research Focus:clustering· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

M. Jefferson· 2 shared T. Wileman· 2 shared Y. Wang· 2 shared A. Kipar· 1 shared A. Mann· 1 shared A. Zhekova· 1 shared B. Bone· 1 shared G. Juergens· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

M. Jefferson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

TECPR1 provides E3-ligase like activity to the ATG5-ATG12 complex to conjugate LC3/ATG8 to damaged lysosomes

Y. Wang, M. Jefferson, J. Mccoll, P. Verkade, U. Mayer, T. Wileman

This study shows TECPR1 can substitute for ATG16L1 to conjugate LC3 to damaged lysosomes by providing E3-ligase activity to the ATG5-ATG12 complex.

bioRxiv · Jun 24, 2023 · PreprintCell Biology

NSF/αSNAP2-mediated cis-SNARE complex disassembly precedes membrane fusion generating the cell plate during Arabidopsis cytokinesis

M. Park, U. Mayer, G. Juergens

The paper identifies NSF/αSNAP2 as the machinery disassembling KNOLLE cis-SNARE complexes during Arabidopsis cytokinesis, preceding cell plate formation.

bioRxiv · Apr 29, 2022 · PreprintPlant Biology

The WD and linker domains of ATG16L1 required for non canonical autophagy limit lethal influenza A virus infection at epithelial surfaces.

Y. Wang, W. Zhang, M. Jefferson, P. Sharma, B. Bone, A. Kipar, J. L. Coombes, T. Pearson, A. Mann, A. Zhekova, Y. Bao, R. A. Tripp, Y. Yamauchi, S. R. Carding, U. Mayer, , T. Wileman

This study shows that non-canonical autophagy, mediated by the WD and linker domains of ATG16L1, protects against lethal influenza A virus infection by limiting viral replication and inflammation in airway epithelial cells.

bioRxiv · Jan 15, 2020 · PreprintMicrobiology