Author

W. E. England

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:bacteria (other)· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

D. E. Campbell· 1 shared D. Luy· 1 shared I. Wickramatunga· 1 shared J. R. Leigh· 1 shared K. C. Tan· 1 shared K. Wong· 1 shared L. T. Doan· 1 shared P. H. Degnan· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

D. E. Campbell

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Interrogation of the integrated mobile genetic elements in gut-associated Bacteroidaceae with a consensus prediction approach

D. E. Campbell, J. R. Leigh, T. Kim, W. E. England, R. Whitaker, P. H. Degnan

The paper introduces VICSIN, a consensus method for predicting and clustering mobile genetic elements in gut-associated Bacteroidaceae, identifying novel MGEs and revealing gene sharing patterns.

bioRxiv · Sep 2, 2021 · PreprintMicrobiology

Isoform-specific aPKC renders primary cilia dispensable for Hedgehog signaling and basal cell carcinoma growth

T. T. Nguyen, K. Wong, U. S. Jeon, V. Jhumkhawala, D. Luy, K. C. Tan, I. Wickramatunga, V. Kumar, W. E. England, L. T. Doan, R. C. Spitale, S. X. Atwood

The paper shows that aPKC isoforms sustain HH signaling and BCC growth without primary cilia by compensating for ciliary loss in advanced cancers.

bioRxiv · Jun 8, 2020 · PreprintCancer Biology