Author

X. Wehrens

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Organism:mouse· 2 Organism:rat· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Bergan-Dahl· 1 shared A. Chong· 1 shared A. Edwards· 1 shared A. P. Quick· 1 shared C. Soeller· 1 shared D. Fraidenraich· 1 shared E. D. Moore· 1 shared E. Himelman· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCell Biology

A. Bergan-Dahl

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Author profile →

4 papers

Recent·Most cited

Junctophilin-2 Regulates Mitochondrial Metabolism

S. Prisco, L. M. Hartweck, F. Kazmirczak, J. B. Mendelson, S. L. Deng, S. K. Lahiri, X. Wehrens, K. Prins

This study shows JPH2 regulates mitochondrial metabolism in the right ventricle, with gene therapy improving RV function in a rat model of RVD.

Circulation · Aug 20, 2024Peer-reviewedPhysiology

PHOSPHORYLATION OF RyR2 SIMULTANEOUSLY EXPANDS THE DYAD AND REARRANGES THE TETRAMERS

P. Asghari, S. Shahrasebi, H. H. Valdivia, X. Wehrens, E. D. Moore

This study examines how phosphorylation of RyR2 affects dyad size and tetramer arrangement in response to isoproterenol using mutant mice.

bioRxiv · May 23, 2023Physiology

Microtubule-Connexin-43 regulation suppresses arrhythmias and fibrosis in Duchenne muscular dystrophy mice.

E. Himelman, J. Nouet, M. Lillo, A. Chong, X. Wehrens, G. Rodney, L. Xie, N. Shirokova, J. E. Contreras, D. Fraidenraich

This study shows that Cx43 phosphorylation status affects microtubule organization in DMD mice, influencing arrhythmias and fibrosis.

bioRxiv · Mar 29, 2022 · PreprintCell Biology

Prolonged β-Adrenergic Stimulation Disperses Ryanodine Receptor Clusters in Cardiomyocytes: Implications for Heart Failure

X. Shen, J. van den Brink, A. Bergan-Dahl, T. R. Kolstad, E. S. Norden, Y. Hou, M. Laasmaa, A. P. Quick, E. K. S. Espe, I. Sjaastad, X. Wehrens, A. Edwards, C. Soeller, W. E. Louch

This study shows prolonged β-adrenergic stimulation causes RyR cluster dispersion in cardiomyocytes, disrupting Ca2+ homeostasis and contributing to heart failure.

bioRxiv · Feb 18, 2022Cell Biology