Author

Y. Anazawa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:C. elegans· 1 Organism:human· 1

Frequent Co-Authors

S. Niwa· 2 shared A. Gennerich· 1 shared A. J. Lam· 1 shared D. W. Nowakowski· 1 shared K. Chiba· 1 shared K. Hayashi· 1 shared K. Okada· 1 shared L. Rao· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

S. Niwa

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2 papers

Recent·Most cited

De novo disease-associated mutations in KIF1A dominant negatively inhibit axonal transport of synaptic vesicle precursors

Y. Anazawa, T. Kita, R. Iguchi, K. Hayashi, S. Niwa

This study establishes C. elegans models for KIF1A-associated neuronal disorder and shows that disease mutations inhibit wild-type KIF1A function in heterodimers.

bioRxiv · Jul 22, 2021Cell Biology

A Highly Conserved 310-Helix Within the Kinesin Motor Domain is Critical for Kinesin Function and Human Health.

A. J. Lam, L. Rao, Y. Anazawa, K. Okada, K. Chiba, S. Niwa, A. Gennerich, D. W. Nowakowski, R. J. McKenney

This study identifies a conserved 310-helix in KIF1A critical for motor function and human health, showing that the P305L mutation disrupts microtubule association and motor performance.

bioRxiv · Sep 22, 2020Biophysics