Author

Y. Elmasry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

C. Cloix· 2 shared G. Ichim· 2 shared A. J. Chalmers· 1 shared A. Koessinger· 1 shared C. Nixon· 1 shared D. Koessinger· 1 shared F. Bock· 1 shared G. Malviya· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

C. Cloix

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Mitochondrial dynamics regulate genome stability via control of caspase-dependent DNA damage

K. Cao, J. S. Riley, C. Cloix, Y. Elmasry, G. Ichim, K. J. Campbell, S. W. Tait

This study shows mitochondrial dysfunction promotes DNA damage through caspase-dependent mechanisms regulated by mitochondrial dynamics.

bioRxiv · Sep 13, 2021Cell Biology

Increased apoptotic priming of glioblastoma enables therapeutic targeting by BH3-mimetics

A. Koessinger, D. Koessinger, K. Kinch, L. Martinez-Escardo, N. Paul, Y. Elmasry, G. Malviya, C. Cloix, K. Campbell, F. Bock, J. O'Prey, K. Stevenson, C. Nixon, M. Jackson, G. Ichim, , S. Tait

This study shows that glioblastoma (GBM) is highly sensitive to BH3-mimetics due to elevated anti-apoptotic BCL-xL and MCL-1 expression, which can be therapeutically targeted.

bioRxiv · Jun 13, 2021 · PreprintCancer Biology