Author

Y. Markaki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:chromatin & epigenetic regulation· 3 Organism:human· 3 Approach:experimental assay· 2 Approach:microscopy & imaging analysis· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

T. Cremer· 2 shared A. Banerjee· 1 shared A. Szczurek· 1 shared B. A. Mistry· 1 shared B. Hübner· 1 shared C. Cremer· 1 shared C. Luong· 1 shared D. Maestrini· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

T. Cremer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

True-to-scale DNA-density maps correlate with major accessibility differences between active and inactive chromatin

M. Gelleri, S.-Y. Chen, A. Szczurek, B. Hübner, M. Sterr, J. Neumann, O. Kröger, F. Sadlo, J. Imhoff, Y. Markaki, M. J. Hendzel, M. Cremer, T. Cremer, H. Strickfaden, C. Cremer

The paper presents true-to-scale DNA-density maps of human and mouse nuclei, revealing significant accessibility differences between active and inactive compartments.

bioRxiv · Mar 23, 2022Cell Biology

Distinctive nuclear zone for RAD51-mediated homologous recombinational DNA repair

Y. Horikoshi, H. Shima, W. Kobayashi, J. Sun, V. J. Schmid, H. Ochiai, L. Shi, F. Atsuhiko, Y. Kinugasa, H. Kurumizaka, T. Ikura, Y. Markaki, S.-i. Tate, K. Igarashi, T. Cremer, S. Tashiro

This study shows RAD51 forms distinctive nuclear zones to facilitate homologous recombination by bridging damaged and intact DNA during repair.

bioRxiv · Nov 29, 2021 · PreprintCell Biology

Xist-seeded nucleation sites form local concentration gradients of silencing proteins to inactivate the X-chromosome

Y. Markaki, J. G. Chong, C. Luong, S. Y. X. Tan, Y. Wang, E. C. Jacobson, D. Maestrini, I. Dror, B. A. Mistry, J. Schoeneberg, A. Banerjee, M. Guttman, T. Chou, K. Plath

This study identifies Xist-seeded nucleation sites that create local concentration gradients of silencing proteins, leading to X-chromosome inactivation through macromolecular crowding.

bioRxiv · Nov 22, 2020Cell Biology