Author

Yan‐Na Lian

Sir Run Run Shaw Hospital

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Li LiuiD· 2 shared Xiang‐Yao LiiD· 2 shared Chen ZhangiD· 1 shared Cheng Wu· 1 shared Han XuiD· 1 shared Hao SuniD· 1 shared Huifang ShangiD· 1 shared Jiajun DongiD· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Li LiuORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Single-cell RNA sequencing uncovers the excitatory/inhibitory synaptic unbalance in the retrosplenial cortex after peripheral nerve injury

Jinghua Wang, Cheng Wu, Yan‐Na Lian, Ziyue Wang, Jiajun Dong, Qin Wu, Li Liu, Li Sun, Wei Chen, Wenjuan Chen, Zhi Zhang, Min Zhuo, Xiang‐Yao Li

The paper uses scRNA-seq to identify cell-type-specific transcriptional changes in the retrosplenial cortex after nerve injury, revealing an excitatory/inhibitory synaptic imbalance linked to pain hypersensitivity.

bioRxiv · Jun 9, 2021 · PreprintNeuroscience

Deconstruction of the retrosplenial granular cortex for social behavior in the mouse model of fragile X syndrome

Huifang Shang, Ruonan Cai, Hao Sun, Tao Sheng, Yan‐Na Lian, Li Liu, Wei Chen, Lixia Gao, Han Xu, Chen Zhang, Jianhong Luo, Xinjian Li, Xiang‐Yao Li

The paper identifies reduced social-related neuronal activity in the retrosplenial granular cortex (RSG) of Fmr1-KO mice, leading to social novelty deficits and suggests RSG manipulation as a potential therapeutic strategy.

bioRxiv · Jan 24, 2021 · PreprintNeuroscience