Author

Yein Chung

University of Toronto

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell-cell communication· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Anthony AltieriiD· 2 shared Erika E. McCartneyiD· 2 shared Matthew B. BuechleriD· 2 shared Akshay T. KrishnamurtyiD· 1 shared Apple Cortez VollmersiD· 1 shared Boris HinziD· 1 shared Christopher D. DavidsoniD· 1 shared Crystal Hu· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

Anthony AltieriORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Bigh3 is essential for pulmonary fibrosis

Anthony Altieri, Yashar Aghazadeh Habashi, Yein Chung, Erika E. McCartney, Ronen Schuster, Mark Abovsky, Faizah S. Abdullah, Shaheed W. Hakim, Sam Cooper, Igor Jurišica, Boris Hinz, Matthew B. Buechler

This study shows Bigh3 is essential for lung fibrosis by demonstrating its role in fibroblast-macrophage interactions and collagen expression.

bioRxiv · Sep 2, 2025 · PreprintImmunology

Dermatopontin-expressing fibroblasts mediate an essential skin macrophage niche

Apple Cortez Vollmers, Sunny Z. Wu, Anthony Altieri, Erika E. McCartney, Hannah S. Bender, Wyne P. Lee, Juan Zhang, Crystal Hu, Salil Uttarwar, Jason A. Vander Heiden, Christopher D. Davidson, Yein Chung, Willie Ortiz, Michael Long, Raymond Asuncion, , Shannon J. Turley

This study identifies Dpt+ fibroblasts as essential for maintaining a Csf1-dependent macrophage niche in skin, with implications for wound healing and scleroderma.

bioRxiv · Nov 21, 2024 · PreprintImmunology