Boris Hinz

This study identifies two distinct macrophage populations in chronic lung allograft dysfunction using single-cell RNA sequencing and characterizes their functional roles in inflammation and fibrosis.

This study identifies EphB4 as a critical mediator of pulmonary fibrosis through genetic and pharmacological approaches, showing reduced ECM gene expression and fibrotic pathology.

This study shows Bigh3 is essential for lung fibrosis by demonstrating its role in fibroblast-macrophage interactions and collagen expression.

This study identifies endomembrane depletion as a key factor in phagocytic capacity exhaustion in macrophages.

This paper develops ultrathin collagen sheets with non-linear tensile behavior, mimicking native tissue properties for biomedical applications.

This paper introduces a method to align single-cell RNA data across species and tissues using adversarial learning, revealing common cell types like macrophages and fibroblasts.

This study identifies MRTF as a key mediator of fibrosis in PKD by showing its activation following PC1/2 loss and its role in epithelial fibrogenesis.