Bijay P. Dhungel

This study investigates the molecular mechanisms of AAV receptor AAVR and AAVR2, identifies functional motifs, and demonstrates enhanced AAV entry through engineered receptors.

The paper identifies unique protein interaction networks in CHD family remodelers by analyzing intrinsically disordered regions and aggregation-prone regions, revealing their role in transcriptional regulation.

The paper shows that chronic myeloid leukemia patients in molecular remission still have widespread aberrant alternative splicing, unlike normal gene expression and DNA methylation patterns.