Magor L. Lörincz

This paper demonstrates that Intersectional Short-Pulse (ISP) stimulation improves seizure control in a rat model of temporal lobe epilepsy compared to conventional TES.

The paper examines cortical neuron firing patterns during absence seizures in awake rodents, finding structured heterogeneity rather than generalized hyperexcitability.

This study identifies a thalamic hub that integrates brainstem and stress signals to regulate REM sleep through activity-dependent modulation of theta oscillations.

The paper analyzes transcriptional burst kinetics in pyramidal and fast-spiking neurons to show that burst parameters differ from average mRNA counts and contribute to neuronal phenotype.

This study shows that closed-loop brain stimulation targeting the amygdala or prefrontal cortex after fear memory reconsolidation reduces fear generalization and enhances extinction in rats.

This study investigates how higher-order thalamic nuclei contribute to the spread and maintenance of spike-and-wave discharges in absence seizures using GAERS models.

This study shows that closed-loop brain stimulation using hippocampal sharp wave-ripples can enhance fear extinction memory consolidation and reduce pathologic fear in a PTSD model.

The paper investigates how serotonin modulates principal neurons and interneurons in the anterior piriform cortex using electrophysiological recordings, optogenetics, and pharmacology, revealing differential suppression and excitation effects.

The paper shows that restoring gamma oscillations in the limbic system via olfactory bulb neuromodulation alleviates depressive-like behaviors in animals.

This study shows that systemic ivabradine, an HCN channel inhibitor, reduces absence seizures in rats, suggesting its potential as a new treatment for this condition.

This study identifies reciprocal GABAergic projections between the lateral hypothalamus and dorsal raphe nucleus that promote wakefulness through mutual inhibition and network activity modulation.

This study shows that LGN neurons in awake mice have arousal-dependent activity linked to pupil size and corticothalamic feedback, forming distinct coalitions between thalamocortical neurons and interneurons.