Author

Guang Lin

Baylor College of MedicineORCID 0000-0001-5594-3397Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Assay:metabolomics· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Akhilesh PandeyiD· 2 shared Hugo Jozef BelleniD· 2 shared Seul Kee ByeoniD· 2 shared Zhongyuan ZuoiD· 2 shared Agnes JH Liang· 1 shared Amanda Hope· 1 shared Benny Abraham KaipparettuiD· 1 shared Burak TepeiD· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersNeuroscience

Akhilesh PandeyORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintApr 14, 2023

Loss of Mitochondrial Enoyl CoA Reductase causes elevated ceramide levels and impairs iron metabolism

Debdeep Dutta, Oguz Kanca, Seul Kee Byeon…·Hugo Jozef Bellen

This study shows that loss of MECR causes elevated ceramide levels and impaired iron metabolism, linking mitochondrial fatty acid synthesis to neurodegeneration.

1 citation

Drosophilaproteomics

𝕏 PostBluesky

NeuroscienceeLifePeer-reviewedJan 16, 2023

Exploring therapeutic strategies for Infantile Neuronal Axonal Dystrophy (INAD/PARK14)

Guang Lin, Burak Tepe, Geoff McGrane…·Hugo Jozef Bellen

The paper identifies ceramide accumulation, lysosomal expansion, and mitochondrial defects as root causes of INAD/PARK14 and tests therapeutic strategies including drugs and gene therapy.

15 citations

mouseDrosophila

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