David M. Raizen

The paper identifies genes modulating stress-induced sleep in C. elegans using automated screening and genetic association analysis.

This study shows that KIN-29 regulates stress-induced sleep in C. elegans via mitochondrial ROS signaling, with ROS acting downstream of KIN-29 and upstream of the ALA neuron.

This study identifies GRK-2 as a regulator of exploration and movement quiescence in C. elegans by acting in chemosensory neurons and opposing neuropeptide signaling pathways.

This study examines pharyngeal pumping defects in C. elegans feeding mutants using high-speed video microscopy to identify specific timing and contraction issues affecting particle transport.

This study shows that dehydrated C. elegans stocks can survive multiple freeze-thaw cycles using trehalose or glycerol.

This study identifies KIN-29, a C. elegans SIK homolog, as essential for metabolic regulation of sleep by linking energy deficits to sleep promotion through nuclear actions in sensory neurons.

The paper identifies a quiescent state in C. elegans, PRQ, which is enhanced by cellular stress and involves neuropeptidergic mechanisms but is not sleep.

This study shows that Caenorhabditis elegans senses and responds to gravity through an active behavior requiring gravisensation, with gravity response declining with age.