Author

Amelia Mazzone

Mayo ClinicORCID 0000-0001-6236-6962Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:statistical model· 3 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Alexandre Gaspar‐MaiaiD· 2 shared Wazim Mohammed IsmailiD· 2 shared Abhilash Sawant· 1 shared Abhishek Avinash MangaonkariD· 1 shared Akhilesh PandeyiD· 1 shared Amik MunankarmyiD· 1 shared Arthur BeyderiD· 1 shared Brooke R. DrulineriD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersGenomics

Alexandre Gaspar‐MaiaORCID

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintSep 7, 2026

Maladaptive Piezo1 Mechanotransduction Drives Smooth Muscle Aging in the Gut

Vikram Joshi, Corey Seavey, Kaitlyn R. Knutson…·Arthur Beyder

This study identifies maladaptive Piezo1 mechanotransduction as a driver of smooth muscle aging in the gut, showing it impairs contractility and contributes to gastrointestinal dysfunction.

DOI 10.64898/2026.09.02.748183

mousebulk RNA-seq

𝕏 PostBluesky

GenomicsbioRxivPreprintApr 16, 2025

Single cell resolution of an epigenetic signature of persister tumor cell

Mihai Gabriel Dumbrava, Wazim Mohammed Ismail, Letícia Sandoval…·Alexandre Gaspar‐Maia

The paper identifies an epigenetic signature associated with persister tumor cells in ovarian cancer that correlates with chemotherapy resistance and defines the persister state.

2 citations

humanscRNA-seq

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GeneticsbioRxivPreprintAug 25, 2022

Single cell multiomic analyses reveal divergent effects of DNMT3A and TET2 mutant clonal hematopoiesis in inflammatory response

Wazim Mohammed Ismail, Jenna A. Fernandez, Moritz Binder…·Mrinal M. Patnaik

This study uses single cell multiomic analyses to show DNMT3A mutations in clonal hematopoiesis worsen inflammatory outcomes in COVID-19 by increasing IL32 expression and chromatin accessibility.

DOI 10.1101/2022.08.25.505316

humanscRNA-seq

𝕏 PostBluesky