Max Löhning

The paper evaluates multi-laboratory studies to identify promising treatments by assessing effect sizes and experimental rigor across different medical fields.

This study shows that TLR activation in OA synovial fibroblasts leads to inflammatory responses and chondrocyte dysfunction.

The paper examines T-bet expression stability in virus-specific Th1 cells during chronic infection, finding that quantitative differences persist and some cells retain Tfh-like features.

This study identifies D-lactate's stereospecific role in enhancing Th1 cell cytotoxicity and inflammation through mitochondrial ROS regulation.

The paper develops a computational framework to infer enhancer-gene networks in T helper cells, revealing T-bet expression intensity as critical for Th cell fate.