Author

Miloš Gojković

University of HelsinkiORCID 0000-0001-6451-9192Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Heidi M. HaikalaiD· 1 shared Iman Khani FarahaniiD· 1 shared Jenna H. RannikkoiD· 1 shared Johannes A. M. MerilahtiiD· 1 shared Jonas BaumgarteniD· 1 shared Kari J. KurppaiD· 1 shared Lynn E. HeasleyiD· 1 shared Maija HollméniD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Heidi M. HaikalaORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintJan 24, 2026

YAP/TEAD drives treatment-induced adaptive immunosuppression in EGFR-mutant lung cancer

Meija Honkanen, Jonas Baumgarten, Nikol Dibus…·Kari J. Kurppa

This study shows that DTP cells in EGFR-mutant lung cancer drive immunosuppression through YAP/TEAD activity, and co-targeting YAP/TEAD improves EGFR TKI therapy efficacy.

DOI 10.64898/2026.01.22.701073

mousehuman

𝕏 PostBluesky