Basel H. Abuaita

The paper shows that prior exposure to immune stimuli enhances protection against MRSA pneumonia by reprogramming alveolar macrophages to acquire neutrophil-derived antimicrobial enzymes.

This study shows that DRP1-mediated mitochondrial fission supports Rickettsia parkeri replication in macrophages by enhancing ATP production and mitochondrial content.

This study shows that activating IRE1α in the lungs during MRSA infection worsens pneumonia by increasing inflammation and cell death, but blocking IRE1α improves survival and reduces lung damage.