Author

Martin W. Lee

Edinburgh Cancer ResearchORCID 0000-0001-6529-5784Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. R. S. BlackiD· 1 shared Ajitha RajaniD· 1 shared Alan SerrelsiD· 1 shared Alexander E.P. LoftusiD· 1 shared Colin SmithiD· 1 shared Emily R. WebbiD· 1 shared Florent GinhouxiD· 1 shared Fraser Laing· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

A. R. S. BlackORCID

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintNov 26, 2025

FAK inhibitor treatment systemically regulates the glioblastoma immune environment via blocking monocyte trafficking

Emily R. Webb, A. R. S. Black, Giovana Carrasco…·Valerie G. Brunton

This study shows that systemic FAK inhibition reduces immunosuppressive macrophages in glioblastoma by blocking monocyte trafficking, enhancing T cell activity and improving survival in a mouse model.

DOI 10.1101/2025.11.26.690731

mousebulk RNA-seq

𝕏 PostBluesky

Developmental BiologyDisease Models & MechanismsPeer-reviewedJan 1, 2021

Resolving the heterogeneity of diaphragmatic mesenchyme: a novel mouse model of congenital diaphragmatic hernia

Louise Cleal, Sophie L. McHaffie, Martin W. Lee…·You-Ying Chau

This study uses mouse models to show that Wt1 deletion in non-muscle mesenchyme of pleuroperitoneal folds causes congenital diaphragmatic hernia by disrupting tissue fusion and cell migration.

13 citations

mouseexperimental assay

𝕏 PostBluesky