Author

Matteo Ferrari

Memorial Sloan Kettering Cancer CenterORCID 0000-0001-6672-3170Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Assay:organoid / primary culture· 1 Organism:yeast· 1

Frequent Co-Authors

Achille PellicioliiD· 1 shared Charles L. SawyersiD· 1 shared Chetan C. RawaliD· 1 shared Emmet Huang-HobbsiD· 1 shared Justin R. LaClairiD· 1 shared Kyrie PappasiD· 1 shared Marco Vincenzo RussoiD· 1 shared Maria JasiniD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMolecular Biology

Achille PellicioliORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 20, 2025

BRCA2 reversion mutation-independent resistance to PARP inhibition in prostate cancer through loss of function perturbations in the DNA pre-replication complex

Kyrie Pappas, Matteo Ferrari, Perianne Smith…·Charles L. Sawyers

This study identifies loss of function mutations in the DNA pre-replication complex as a mechanism for PARP inhibitor resistance in BRCA-mutant prostate cancer, independent of reversion mutations.

1 citation

mouseorganoid / primary culture

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Molecular BiologyNature CommunicationsPeer-reviewedJun 23, 2020

Rad9/53BP1 promotes crossover recombination DNA repair by limiting the Sgs1 and Mph1 helicases

Matteo Ferrari, Chetan C. Rawal, Samuele Lodovichi…·Achille Pellicioli

The paper shows Rad9 limits helicase recruitment to DNA breaks, promoting specific recombination pathways and reducing genome instability.

30 citations

yeastexperimental assay

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