Michael P. Murrell

The paper shows that energy dissipation in active actomyosin networks depends on mechanical state rather than motor abundance, revealing degenerate dissipation through state-dependent energy conversion.

The paper shows that a mechanical tension gradient encoded by tissue geometry drives epithelial renewal and cell movement in the intestine.

The paper shows how caging of MCA proteins can amplify changes in membrane viscosity and protein length, leading to symmetry breaking in cellular mechanics.

The paper measures energy partitioning between chemical and mechanical subsystems in the cell cortex and finds that energy distribution changes with Rho GTPase pathway manipulation.

This study develops a model showing that rapid actin filament growth leads to kinetic trapping of bent filaments, influenced by subunit availability and crosslinking.

The paper identifies a molecular clock mechanism that regulates cell migration in confined spaces through cyclic RhoA activity driven by Ca2+ and GEF-H1 oscillations.