Feng He

The paper examines stress granule dynamics and transcriptome changes under glycolytic inhibition, finding that OXPHOS transcripts are sequestered in SGs, leading to mitochondrial dysfunction and apoptosis.

This study reveals that p21 continues to play a role in cell cycle re-entry after G0/G1 arrest, beyond its initial inhibitory function.

This study uses a zebrafish model of Feingold syndrome 1 to show that Mycn regulates intestinal development through ribosomal biogenesis and that leucine supplementation can rescue the intestinal defect.