Author

Wei Wu

Indiana University IndianapolisORCID 0000-0002-7835-4963Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Wei Wu4 papers · Army Medical University

Biology Disciplines

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Neuroscience· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1 Organism:rat· 1

Frequent Co-Authors

Chen CheniD· 2 shared Xiangbing WuiD· 2 shared Xiao‐Ming XuiD· 2 shared Xiaolong Du· 2 shared Alan Yaacoub· 1 shared Christopher B. Shields· 1 shared Christopher H. Yu· 1 shared Chun‐Li ZhangiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersPathology

Chen ChenORCID

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Author profile →

3 papers

Recent·Most cited

Early blood-spinal cord barrier stabilization preserves peri-lesional tissue and improves recovery after SCI

Chen Chen, Yan Sun, Xiaolong Du, Seung Young Lee, Xiaoting Wang, Qi Han, Wenhui Xiong, Xiangbing Wu, Yiping Zhang, Christopher B. Shields, Xiaoming Jin, Ji‐Xin Cheng, Wei Wu

This study identifies early vascular leakage beyond the injury epicenter as a driver of secondary pathology after SCI and shows that stabilizing the blood-spinal cord barrier improves recovery.

bioRxiv · Dec 7, 2025 · PreprintPathology

NG2 Glia Reprogramming Induces Robust Axonal Regeneration After Spinal Cord Injury

Wenjiao Tai, Xiaolong Du, Chen Chen, Xiao‐Ming Xu, Chun‐Li Zhang, Wei Wu

This study shows that reprogramming NG2 glia after spinal cord injury leads to axonal regeneration and improved function.

iScience · Jan 19, 2024Peer-reviewedNeuroscience

CD1d-dependent neuroinflammation impairs tissue repair and functional recovery following a spinal cord injury

Xiangbing Wu, Jianyun Liu, Wei Li, Mohammad Faizan Khan, Heqiao Dai, Jeremy Tian, Raj Priya, Daniel J. Tian, Wei Wu, Alan Yaacoub, Jun Gu, Fahim Syed, Christopher H. Yu, Xiang Gao, Qigui Yu, , Randy Richard Brutkiewicz

This study shows CD1d promotes proinflammatory responses after spinal cord injury, impairing tissue repair and functional recovery, and identifies CD1d as a potential therapeutic target.

bioRxiv · Oct 13, 2023 · PreprintNeuroscience